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Dysphasia is a partial or complete impairment of the ability to communicate resulting from brain injury. Approximately one million Americans currently have one of the various forms of dysphasia, and an additional 80,000 new cases occur annually. The term "dysphasia" is more frequently used by European health professionals, whereas in North American the term "aphasia" is more commonly preferred. These two terms, however, can be and are used interchangeably. They both refer to the full or partial loss of verbal communication skills due to damage or degeneration of the brain's language centers. Developmental dysphasia is considered to be a learning disability, but will not be the focus of this article. Verbal communication is derived from several regions located in the language-dominant hemisphere of the brain. These include the adjacent inferior parietal lobe, the inferolateral lobe, and the poster-osuperior temporal lobe, as well as the subcortical connection between these areas. Disease, direct trauma, lesion, or infarction involving one or more of these regions can disrupt or prevent proper language function. Dysphasia does not necessarily prevent proper cognitive function, so the patient can think and feel with perfect clarity. This can be extremely frustrating for the patient, as they cannot express these thoughts and feelings to others. Dysphasia can occur in a variety of forms, depending on how the communicative disruption manifests. Classically, dysphasia can affect one or more of the basic language functions: comprehension (understanding spoken language), naming (identifying items with words), repetition (repeating words or phrases), and speech. Although there are several subtypes of dysphasias, they most commonly manifest in one of three syndromes: expressive dysphasia, receptive dysphasia, or global dysphasia. Expressive dysphasia, also known as motor dysphasia, produces a conscious and recognizable disruption of a patient's speech production and language output. This includes the impairment of speech initiation, proper grammatical sequencing, and proper word forming and articulation. Although patients can perfectly understand what is said to them, they have great difficulty communicating their thoughts. Broca's dysphasia is the most common type of expressive dysphasia. It is caused by damage to the lower area of the premotor cortex, located just in front of the primary motor cortex. This region is most commonly referred to as the Broca's area. Speech for patients with Broca's dysphasia may be completely impossible. Others may be able to form single words or full sentences, but only through great effort. "Telegraphing," the omission of articles and conjunctions, may also be exhibited. Also known as isolation syndrome, transcortical dysphasia is caused by damage to the language-dominant brain that separates all or parts of the central region from the rest of the brain. There are three sub-classes of transcortical dysphasia, which define the impairments to a patient's ability to repeat words, sentences, and phrases: transcortical motor dysphasia, transcortical sensory dysphasia, and mixed transcortical dysphasia. Additional impairments may occur depending on the extent and location of the damage.Receptive dysphasia, also known as sensory dysphasia, impairs the patient's comprehension and meaning of language. Unlike expressive dysphasia, the patient can speak fluently and articulately, but will utilize meaningless words, nonsensical grammar, and unnecessary phrases to the point of becoming incomprehensible. However, they will be completely unaware of their mistakes. Additionally, the patient will find it difficult to comprehend spoken language and/or word-object relation.
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